Klerimed is a semisynthetic macrolide antibiotic, structurally very similar to erythromycin. It inhibits protein synthesis by penetrating the cell wall and binding to the 50S ribosomal subunit. The absolute bioavailability of Klerimed after oral administration is 50-55%, which probably underestimates its systemic activity, as it undergoes extensive first pass metabolism to its active metabolite, 14-hydroxyKlerimed. Limited data are available on the distribution of Klerimed in humans. It appears to be distributed into most body tissues and fluids. However, no data is available on CSF penetration of Klerimed. Elimination of Klerimed follows nonlinear kinetics. It is extensively metabolized in the liver and excreted as parent drug and metabolites in urine, and faeces.
Other names for this medication:
Clarithromycin,
Biaxin,
Abbotic,
Aeroxina,
Biclar,
Binoclar,
Bremon,
Clabel,
Clamycin,
Clarimac,
Clarimax,
Clarimed,
Clarimycin,
Claripen,
Claritt,
Clariva,
Clariwin,
Clarix,
Claromycin,
Claron,
Cleron,
Emimycin,
Euromicina,
Finasept,
Fromilid,
Infex,
Kailasa,
Kalixocin,
Klabax,
Klabet,
Klabion,
Klarimac,
Klarimax,
Klarithran,
Klax,
Klaz,
Klerimed,
Kofron,
Krobicin,
Lekoklar,
Macladin,
Maclar,
Macrobid,
Macrol,
Monozeclar,
Naxy,
Synclar,
Taclar,
Veclam,
Vikrol,
Xylar,
Zeclar,
Zeclaren,
Similar Products:
Caverject,
Normax,
Elbasvir,
Zoraxin,
Neksium,
Flemoxin,
Ticamet,
Uroxal,
Metazole,
Norsic,
Penmox,
Levofloxacinum,
Rhinolast,
Clomipramine,
Ezol,
Ritonavir,
Gastrazol,
Lipophage,
Azitrocin,
Uzol,
Clamycin,
Dapsone,
Flubiotic,
Klax,
Gordius,
Elopram,
Osiren,
Trimezol,
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Prevacid,
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Ciplin,
Aropax,
Anafranil,
Duzimicin,
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Mutations in alpha6 or beta4 integrins (ITGA6, ITGB4) are known to cause junctional epidermolysis bullosa with pyloric atresia (JEB-PA), often lethal in infancy through skin desquamation. We compared the survival of patients who had return of spontaneous beating (ROSB) after novamin prescription toothpaste ECPR with the survival of those who had return of spontaneous circulation (ROSC) after conventional CPR. During the course of sepsis, endotoxins and cytokines activate Kupffer cells, induce the liberation and synthesis of adhesion molecules, and damage hepatocytes, which leads to septic liver failure.
An improved parasitological technique for the diagnosis of African trypanosomiasis? The data suggest that the nurse manager role will continue to increase in complexity and importance. Supplementation with bacterial isolates belonging to the Firmicutes phylum abrogated the heightened TH17 responses in vitro. Bacillus subtilis lon protease lomac 430 usa today sudoku prevents inappropriate transcription of genes under the control of the sporulation transcription factor sigma G.
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