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Virol (Abacavir)
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Abacavir

Virol is an antiretroviral medicine. It is used for human immunodeficiency virus (HIV) infection. It slows the progress of HIV infection, but it is not a cure. HIV destroys cells in the body, called CD4 T cells. These cells are a type of white blood cell and are important because they are involved in protecting your body from infection. If left untreated, the HIV infection weakens your immune system so that your body cannot defend itself against bacteria, viruses and other germs. Virol slows down the progress of HIV infection by reducing the amount of virus in your body. It does this by stopping the virus from copying (replicating) itself.

Other names for this medication:
Abacavir, Abalam, Epzicom, Kivexa, Abamune, Virol, Ziagen,

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Here we show that a feedback cavity architecture breaks the harmonic-oscillator restriction. Axial loading of the spine during CT and MR in patients with suspected lumbar spinal stenosis. We establish liver metastasis model of human pancreatic cancer cells in Balb/c nude mice and find that knocking down Par3 promotes invasion and metastasis and disturbs tight junction assembly in vivo. Metric properties and normative data for brief noise and electromagnetic field sensitivity scales.

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To assess the prognostic value of biologic (p53, Ki-67) and clinical factors in squamous cell carcinoma of the oropharynx after radical surgery and postoperative radiotherapy (RT). Chronologic alteration of suppressive effects of serum from liver-grafted rats against graft versus host disease. A rat olfactory epithelium cDNA library was screened for olfactory receptor clones.

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Synthetic analogs of vitamin D3 have inhibitory effects on breast cancer cell lines. Feasibility of an electronic participant information system for adult day services: A qualitative study of staff perspectives. Relationship of tissue factor pathway inhibitor activity to plasma lipoproteins and myocardial infarction at a young age. The aim of this study was to determine the association between family supports in the postpartum period with occurrence of post-traumatic stress disorder following preeclampsia. Results from direct real-time visualization of blood flow studies demonstrate activation-independent platelet aggregation, which occurs exclusively through binding of soluble VWF to platelet GPIb at very high shear stress, and may thus contribute to acute thrombotic occlusion.

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Recent advances, combining structural biology, cryo-electron tomography, molecular genetics, in vivo imaging, bioinformatics and biophysics, have greatly increased our understanding of the T3SS, especially the structure of its transmembrane and cytosolic components, the transcriptional, post-transcriptional and functional regulation and the remarkable adaptivity of the system. A comparative review of oxybutynin chloride formulations: pharmacokinetics and therapeutic efficacy in overactive bladder.

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